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Summary Immune Modulation Of Learning, Memory, Neural Plasticity And Neurogenesis

Course
- Scriptie
- Yannick Mahieu
- 2016 - 2017
- Vrije Universiteit Amsterdam
- _TEACHER_GENERAL_
36 Flashcards & Notes
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A snapshot of the summary - Immune modulation of learning, memory, neural plasticity and neurogenesis

  • 1 The role of the immune system in learning, memory, neural plasticity and neurogensis under quiescent conditions

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  • What is the mayor function of the first chapter?
    To review the physiological beneficial effects of the immune system under normal, quiescent conditions.
  • Does the immune system function autonomously?
    No, the immune system is influenced by the endocrine and the neural system too.
  • Which immune cells do interact with the brain to aid in neural plasticity?
    • microglia
    • astrocytes
    • dendritic cells
    • T cells
    • endothilial cells
  • Which immune systems are involved in synaptic pruning?
    • The MHC-1
    • the complement system
  • How can the brain activate the endocrine system?
    By neural stimulation of the HPA-axis and the autonomic nervous system.
  • Why did the interaction between the immune system and the brain under normal circumstances not receive attention?
    Probably because of the overwhelming evidence for brain inflammation caused by immune processes.
  • 1.1.1 The role of T cells

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  • What is the main idea about T CD4 cells directed to self-antigens?
    The are thought to be neuroprotective.
  • Who postulated that circulating T cells play a supportive role in brain and mind functioning?
    Jonathan Kipnis and Michaela Schwartz
  • What is Kipnis and Scwartz their claim that T cells are involved in neuroplasticity?
    T cell deficient mice exhibit impaired hippocampal learning and memory.
  • How did Kipnis (2004) restore learning deficits induced by neurotransmitter abnormalities?
    He boosted the T-cells of the animals with a weak agonist of auto-antigen T cells.
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