Anorexia, obesity, and diabetes
13 important questions on Anorexia, obesity, and diabetes
What is produced when glucose drops?
Diabetes mellitus type 1 resembles fasting. What happens?
What are the consequences of glucagon release?
- Mobilisation of proteins/AA in myocytes
- Mobilisation of FA from adipocytes
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What is so remarkable about patients with diabetes type 1?
- Their glucose is very high
- Their ketone body levels are very high
- Their urea secretion is very high
- Their insulin levels are really low
- Their glucagon levels are really high
What is the path to a keto-acidosis death for diabetic patients?
What is the function of ubiquitin?
What is Atrogin-1 (muscle atrophy F-box)?
Why does the mortality rate increase for people with obesity?
What is the difference between pre-DM2 and DM2?
DM2 gives no glucose homeostasis, despite (high) insulin.
What is the end state of DM2?
What is indicative of long term exposure to hyperglycemia?
Can we overrule hormonal regulation?
- AMP inhibits gluconeogenesis and stimulates glycolysis by promoting glucose transportes (GLUT4) to go to the membrane and take up glucose
- Drug treatment: metformin stimulates AMP-dependent kinase
- This treatment is anti-obesity and anti-diabetic. The side effect is that you become more sensitive to insulin to brain gets signals to eat more
What do you know about malonyl-CoA as signaling molecule?
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